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In a mouse model of streptozotocin-induced diabetes with hyperglycemia, high glucose not only increased neutrophil infiltration but also impaired the activity of peroxisome proliferator-activated receptor (PPAR), a transcription factor for lactoferrin (Ltf) encoded by the PPAR gene

D.HuangW.et al (2007)

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Monotherapy reached near 10.8 percent at the top dose by week 26 against roughly 3 percent on placebo, with the usual mild appetite-agent side effects