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Notably, preclinical data characterize this receptor activation as remarkably ligand-efficient, with biologically relevant responses observed at very low ligand concentrations across in vitro and in vivo preclinical systems

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Activation of these receptors appears to reduce the firing rate of dopaminergic neurons in response to reward cues, decreasing anticipatory dopamine release that underlies craving and motivation to seek rewards

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cGAS surveillance of micronuclei links genome instability to innate immunity