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In contrast to the HP meal, the MUO subgroup and MHL subgroup seems to be heading towards the opposite direction
We anticipate that the development of new technologies will similarly facilitate the examination of intracellular GLUT4 trafficking stages such as sorting into GSVs a step that may be impaired in insulin resistance (Figure 3)
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mice lacking prolactin receptors showed a significant reduction in adipose tissue mass and the number of adipocytes ( in vitro experiments have shown that prolactin receptor (PRLR) gene expression is significantly upregulated during the differentiation of preadipocytes into mature adipocytes, with an approximately 90-fold increase, supporting the functional role of prolactin signaling in adipocyte development and lipid accumulation ( In rodent models, pathological hyperprolactinemia also significantly affects brown adipose tissue (BAT), manifested as the downregulation of thermogenic markers, BAT whitening, and impaired adaptive thermogenesis
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