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Studies have found that impaired brain insulin-PI3K-AKT signaling may promote neurodegeneration in Alzheimers disease (AD) by downregulating O-GlcNAcylation, subsequently promoting abnormal tau hyperphosphorylation and neurofibrillary degeneration (Liu et al., 2011b)

Critically, it does NOT activate growth hormone receptors or stimulate IGF-1 signaling, which distinguishes it from full-length human growth hormone (hGH) and theoretically reduces the incidence of systemic growth-promoting effects

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Recently, Hwangbo et al