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Direct Evidence for A Role of Redox Dysregulation in Als In this section, we discuss evidence demonstrating that the central cellular redox regulatory mechanisms and associated proteins are perturbed in ALS (Table 2)

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It was found that m6A modification-associated protein (IGF2BP3), which is highly expressed in human cervical cancer, can enhance Gln metabolism, promote lactate production and secretion, and affect Treg cell differentiation by up-regulating GLS and GLUD1, thus leading to immune escape [96]

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