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ALC acts neuroprotectively by improving the energetic function of brain mitochondria, the elimination of oxidative products, the stabilization of the cell membranes and neurotrophic factor production (stimulates protein and phospholipid biosynthesis), and it exerts anti-apoptotic functions, as well as modulating the expression of genes coding proteins, and protects neural cells from excitotoxicity [73]

The NO/VEGFR2/Akt-eNOS Pathway One of the most frequently cited mechanisms involves the nitric oxide (NO) signaling pathway and vascular endothelial growth factor receptor 2 (VEGFR2)

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