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The increasing level of mitochondrial acetyl-CoA, as a result of hepatic fatty acid oxidation, results in increased transport of citrate to the cytosol which allosterically inhibits PFK-1

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Shows the highest GLUT4 translocation and IGF-1R sensitivity

Its primary pharmacological characterization was published in European Journal of Endocrinology (PMID 9849822), where the authors identified it as the first growth-hormone-secretagogue receptor (GHSR-1a) agonist achieving GH-release selectivity comparable to GHRH in animal models

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